Information on early abortion procedures 2
- Information on early abortion procedures 2
i) The Manual Vacuum Aspiration (MVA)
Procedure and the Abortion Pill (different from a Morning-after Pill) are early abortion methods. The World Health Organization (WHO) approves them as the safest, for before 10 weeks. Women may be told that an early pregnancy comprises i) a thickened tissue, lining the uterus (like what develops before menstruation) and ii) a grape-sized fluid-filled gestational sac. They will not be told that this sac contains their baby.
Manual Vacuum Aspiration (MVA) detaches the womb’s lining from the wall of the uterus. A hand-held instrument is used to remove the tissue and sac. A local anesthetic is usually taken. This procedure avoids invasive surgery (as with late abortion) and scraping out of the womb. MVA is completed in 5 minutes, has a higher success rate than the Abortion Pill and there is much less bleeding and cramping. MVA takes 15 minutes from the start to full recovery.
1. ii) The Abortion Pill causes ‘medical abortion’. Dr. Étienne-Émile Baulieu, in 1980, discovered a synthetic steroid, Mifepristone (the Abortion Pill), an anti-progestin which blocks the action of progesterone, a hormonenecessary to maintain a pregnancy. The Roussel-Uclaf company patented the drug as RU-486 (Mifeprex in the US). Without progesterone, the uterus lining breaks down, with some bleeding. If (24-72 hrs later) a Misoprostol pill is also taken, period-like cramps and more bleeding follow. This drug makes the uterus contract, crushing and expelling the baby, who will be dead in 95-99% of cases. Time off work is not usually needed.
Other early and late abortion methods exist. Methotrexate disables the life support systems of the baby, again expelled with Misoprostol. In most cases, a successful ‘medical abortion’ will cause a feeling like that of a miscarriage. Along with heavy bleeding and cramping, there may be nausea, dizziness, abdominal pain, chills, mild feverishness and diarrhoea. Prostaglandin hormones, injected into the amniotic sac (an ‘instillation’ method), bring on early labour. The fœtus is usually crushed to death in the delivery. Salt poisoning in the amniotic sac kills by the way of dehydration, convulsions and brain hemorrhaging. The skin of the fœtus is burned and blistered, forming a hideous mess. At least 113 ‘instillation’ abortions (which include saline) were performed in the US in 2013. Showing gruesome abortion pictures is criticised. Where a mentality of no-limits, wild savagery is indicated, in getting rid of unwanted babies, clinical examination both of this mentality and of how it is met by society is warranted.
- https://www.earlyabortion.com/abortion-methods/www.newkidscenter.com/Abortion-Pills.html https://www.bpas.org/abortion-care/abortion-treatments/the-abortion-pill/
iii) ‘Morning-after Pill’ is an inaccurate term. The pills comprise different drugs, taken after unprotected sex. The pills are effectively the same as oral contraceptive pills, with a higher hormonal dosage. They must be started as soon as possible – not the ‘morning after’ and generally not later than 72 hrs after sex.
There are two types of morning-after pill: Levonelle and ellaOne. Levonelle contains levonorgestrel (Plan B in the US), a synthetic version of the hormone progesterone. ellaOne contains ulipristal acetate, which stops progesterone from working normally and can delay egg release from the ovary. Levonelle and ellaOne are available in Ireland without prescription.
Manufacturers and providers claim these pills prevent or delay ovulation and are not abortifacient. The complex mechanisms of action (MOAs) of these are, however, not well known. Companies call ‘Morning-After Pills’‘Emergency Contraception’ – downplaying whether they are life-ending or life preventing.
- https://onlinedoctor.lloydspharmacy.com/uk/morning-after pill
iv) The National Center for Biotechnology Information (NCBI), part of the United States National Library of Medicine (NLM) - a branch of the National Institutes of Health (NIH) – houses details on Emergency Contraception (referred to below), which are not published by the manufacturers of these drugs nor by bodies which promote the abortion agenda. It is evident that such organisations, at various levels of influence, side-step or do not engage with information which defines EC pills as abortifacients under certain or all circumstances.
Levonorgestrel for Emergency Contraception (LNG-EC) - likely an Abortifacient Articles eligible for this review were first identified by electronic means, with 2,425 titles retrieved. LNG is a synthetic progestin, contained in oral contraceptive pills and widely used. As one 1.5mg dose or two 0.75mg doses, 12 hrs apart, it is taken within 72 hrs (or 120 hrs at most) of unsafe sex. Sperm can wait up to 120 hrs, in the cervical crypts or fallopian tubes, to fertilize an ovum.
The International Consortium for Emergency Contraception (ICEC) and the International Federation of Gynecology & Obstetrics (FIGO) in 2008, 2011, and again 2012, asserted that “inhibition or delay of ovulation is LNG-EC pills’ principal and possibly only MOA.” The American College of Obstetrics and Gynecology (ACOG) have repeated the claim. The FDA, however, says LNG-EC mainly prevents ovulation or fertilisation (by altering tubal transport of sperm and/or ova) but may also inhibit implantation (by altering the endometrium). Authorities often try to create fake facts.
Laboratory studies report that LNG, at usual doses for EC, has little or no effect upon cervical mucus or the endometrial environment – both affecting sperm activity. However, an insufficient corpus luteum function occurs regularly, in most women who ovulate after taking LNG-EC pills, in pre-ovulatory days. These are the most fertile in the cycle, in which most intercourse occurs and fertilisation most likely. The luteal effects (such as such as lowered progesterone, LH, glycodelin levels, shortened luteal phase and altered endometrial histology) suggest a pre-ovulatory induced post-fertilisation effect. The question of pre-ovulatory LNG-EC use, leading to post-ovulatory changes - on endometrial receptivity and implantation - is yet to be examined. It could show apost-fertilisation LNG-EC MOA.
As noted, published scientific information relating to EC drugs (and to LNG-EC in particular), has repeatedly insisted that the main MOA of these drugs, if not the only one, is to inhibit or delay ovulation. Therapeutic guidelines for family planning, provided by scientific societies, have also stated that there are no post-fertilisation effects to raise ethical objections to the use of LNG-EC.
The examination of reported findings in the literature, however, strongly suggests that pre-fertilisation activity of the drug could, in a routine clinical setting, prevent less than 15 % of expected conceptions - and that pre-ovulatory drug administration can lead to the post-fertilisation luteal effects. LNG-EC administration, during the pre-ovulatory days, cannot prevent ovulation or fertilisation, with a dominant pre-fertilisation MOA, but can be demonstrated to impair luteal function, adversely affecting the survival of the embryo.